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Additionally, ROS can induce autophagy-mediated cell death through various means, such as regulating autophagy-related gene 4 (ATG4) activity and activating the Adenosine Monophosphate-activated protein kinase (AMPK), ataxia-telangiectasia mutated (ATM) and checkpoint kinase 2 (CHK2) pathways [54–58]

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Conclusion: Grx2’s anti-apoptotic and cytoprotective roles may make it a potential drug target to treat oxidative stress-induced diseases Despite being overlooked, research throughout the years has portrayed Grx2 as a capable and robust antioxidant enzyme through direct detoxification of free radicals, cytoprotection, and anti-apoptotic abilities [5, 23]